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NPAS2 dampens chemo-sensitivity of lung adenocarcinoma cells by enhancing DNA damage repair - Cell Death & Disease
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Chemotherapeutic agents, including cisplatin, have remained a cornerstone of lung adenocarcinoma (LUAD) treatment and continue to play an essential role in clinical practice, despite remarkable progress in therapeutic strategies. Hence, a thorough comprehension of the molecular mechanisms underlying chemotherapeutic agent resistance is paramount.
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